Investigation Report on Universal Multimedia Access

Universal Multimedia Access (UMA) refers to the ability to access by any user to the desired multimedia content(s) over any type of network with any device from anywhere and anytime. UMA is a key framework for multimedia content delivery service using metadata. This investigation report analyzes the state-of-the-art technologies in UMA and tries to identify the key issues of UMA. The state-of-the-art in multimedia content adaptation, an overview of the standards that supports the UMA framework, potential privacy problems in UMA systems and some new UMA applications are presented in this report. This report also provides challenges that still remain to be resolved in UMA to make clear the potential key problems in UMA and determine which ones to solve

New Frontiers in Universal Multimedia Access

Universal Multimedia Access (UMA) refers to the ability to access by any user to the desired multimedia content(s) over any type of network with any device from anywhere and anytime. UMA is a key framework for multimedia content delivery service using metadata. This report consists of three parts. The first part of this report analyzes the state-of-the-art technologies in UMA, identifies the key issues and gives what are the new challenges that still remain to be resolved in UMA. The key issues in UMA include the adaptation of multimedia contents to bridge the gap between content creation and consuming, standardized metadata description that facilitates the adaptation (e.g. MPEG-7, MPEG-21 DIA, CC/PP), and UMA system designing considering its target application. The second part introduces our approach towards these challenges; how to jointly adapt multimedia contents including different modalities and balance their presentation in an optimal way. A scheme for adapting audiovisual contents and its metadata (text) to any screen is proposed to provide the best experience in browsing the desired content. The adaptation process is modeled as an optimization problem of the total value of the content provided to the user. The total content value is optimized by jointly controlling the balance between video and metadata presentation, the transformation of the video content, and the amount of the metadata to be presented. Experimental results show that the proposed adaptation scheme enables users to browse audiovisual contents with their metadata optimized to the screen size of their devices. The last part reports some potential UMA applications especially focusing on a universal access application to TV news archives as an example

Balancing Video and Metadata Adaptation for Universal Multimedia Access

This paper presents a framework for joint adaptation of an audiovisual content and its metadata. The presentation of the audiovisual content and its metadata are balanced to fit the given screen size in a way that maximizes user experience in browsing the desired content. The adaptation process is modeled as an optimization problem of the total value of the content provided to the user. The total content value is maximized by jointly controlling the balance between video and metadata presentation, the adaptation way of the video content, and the quantity and quality of metadata to be presented considering the device screen size and the browsing preferences of the user. Experimental results show that this scheme enables users to browse audiovisual contents with their metadata optimized to the screen size of their devices. A demonstration using this scheme is available on http://itswww.epfl.ch/ eiji/umademo

The InVID Plug-in: Web Video Verification on the Browser

This paper presents a novel open-source browser plug-in that aims at supporting journalists and news professionals in their efforts to verify user-generated video. The plug-in, which is the result of an iterative design thinking methodology, brings together a number of sophisticated multimedia analysis components and third party services, with the goal of speeding up established verification workflows and making it easy for journalists to access the results of different services that were previously used as standalone tools. The tool has been downloaded several hundreds of times and is currently used by journalists worldwide, after being tested by Agence France Presse (AFP) and Deutsche Welle (DW) journalists and media researchers for a few months. The tool has already helped debunk a number of fake videos

Influenza Virus Infection Induces Metallothionein Gene Expression in the Mouse Liver and Lung by Overlapping but Distinct Molecular Mechanisms

Metallothionein I (MT-I) and MT-II have been implicated in the protection of cells against reactive oxygen species (ROS), heavy metals, and a variety of pathological and environmental stressors. Here, we show a robust increase in MT-I/MT-II mRNA level and MT proteins in the livers and lungs of C57BL/6 mice exposed to the influenza A/PR8 virus that infects the upper respiratory tract and lungs. Interleukin-6 (IL-6) had a pronounced effect on the induction of these genes in the liver but not the lung. Treatment of the animals with RU-486, a glucocorticoid receptor antagonist, inhibited induction of MT-I/MT-II in both liver and lung, revealing a direct role of glucocorticoid that is increased upon infection in this induction process. In vivo genomic footprinting (IVGF) analysis demonstrated involvement of almost all metal response elements, major late transcription factor/antioxidant response element (MLTF/ARE), the STAT3 binding site on the MT-I upstream promoter, and the glucocorticoid responsive element (GRE1), located upstream of the MT-II gene, in the induction process in the liver and lung. In the lung, inducible footprinting was also identified at a unique gamma interferon (IFN-γ) response element (γ-IRE) and at Sp1 sites. The mobility shift analysis showed activation of STAT3 and the glucocorticoid receptor in the liver and lung nuclear extracts, which was consistent with the IVGF data. Analysis of the newly synthesized mRNA for cytokines in the infected lung by real-time PCR showed a robust increase in the levels of IL-10 and IFN-γ mRNA that can activate STAT3 and STAT1, respectively. A STAT1-containing complex that binds to the γ-IRE in vitro was activated in the infected lung. No major change in MLTF/ARE DNA binding activity in the liver and lung occurred after infection. These results have demonstrated that MT-I and MT-II can be induced robustly in the liver and lung following experimental influenza virus infection by overlapping but distinct molecular mechanisms